Opinion

Kimberly Clark Corp. v. Workers' Compensation Appeal Board

  • 161 A.3d 446
  • 2017 Pa. Commw. LEXIS 175
  • 2017 WL 1739718
Court
Commonwealth Court of Pennsylvania
Filed
May 4, 2017
Status
Published
Author
Covey
On the bench
Leavitt, Covey, Pellegrini
Cited by
18 cases
Authority
More cited than 74.5%

The opinion

IN THE COMMONWEALTH COURT OF PENNSYLVANIA

Kimberly Clark Corporation, :

Petitioner :

:

v. :

:

Workers’ Compensation Appeal :

Board (Bromley), : No. 656 C.D. 2016

Respondent : Submitted: October 14, 2016

BEFORE: HONORABLE MARY HANNAH LEAVITT, President Judge

HONORABLE ANNE E. COVEY, Judge

HONORABLE DAN PELLEGRINI, Senior Judge

OPINION BY

JUDGE COVEY FILED: May 4, 2017

Kimberly Clark Corporation (Employer) petitions this Court for review

of the Workers’ Compensation (WC) Appeal Board’s (Board) March 30, 2016 order

affirming the Workers’ Compensation Judge’s (WCJ) decision granting Sharon R.

Bromley’s (Claimant) Fatal Claim Petition for Compensation by Dependents of

Deceased Employees (Fatal Claim Petition). Essentially, there are two issues before

this Court: (1) whether Claimant met her burden under Section 301(c)(1) of the WC

Act (Act)1 of proving that her deceased husband Donald J. Bromley’s (Bromley)

injury and/or death were caused by exposure to chemicals in Employer’s workplace

and, (2) whether the WCJ issued a reasoned decision.2 After review, we affirm.

1

Act of June 2, 1915, P.L. 736, as amended, 77 P.S. § 411(1).

2

Employer presented six issues for this Court’s review: (1) whether Claimant met her

burden under Section 301(c)(1) of the Act of proving that Bromley’s injury and/or death were

caused by exposure to chemicals in Employer’s workplace; (2) whether Claimant sustained her

factual burden of proving that Bromley was exposed to various workplace chemicals, and that such

exposure rose to the level of a hazard; (3) whether Claimant sustained her burden under Section

301(c)(1) of the Act of proving medical causation between Bromley’s workplace exposure and his

injury and/or death; (4) whether the WCJ erred by overruling Employer’s objection during Barry L.

Singer, M.D.’s June 25, 2010 deposition; (5) whether Claimant met her burden under Section

301(c)(1) of the Act of proving Bromley’s exposure within 300 weeks of his injury and/or death;

Employer operates a paper manufacturing business which produces

napkins, toilet tissue and paper towels. See Reproduced Record (R.R.) at 12a-13a.

Bromley was employed as one of Employer’s Chester plant electricians from 1973 to

2005, during which time he was exposed to various chemicals used in Employer’s

production. In the summer of 2005, Bromley was diagnosed with metastatic bladder

cancer which caused his death on June 23, 2006.

On August 4, 2008, Claimant filed a Claim Petition for WC (Claim

Petition), seeking lost wages from August 11, 2005 through June 23, 2006, plus

medical benefits and counsel fees arising from Bromley’s work injury, which was

described as bladder cancer, multiple pulmonary metastatis and asbestos-related

pleural plaques. See Certified Record (C.R.) Item 4. On that same date, Claimant

filed a Fatal Claim Petition for Compensation By Dependents For Death Resulting

From Occupational Disease (Fatal OD Claim Petition) under Section 301(c)(2) of the

Act, alleging that Bromley’s death was due to an occupational disease - metastatic

bladder cancer - caused by exposure to carcinogens during the course and scope of

his employment with Employer. See C.R. Item 1. Claimant also filed the Fatal

Claim Petition pursuant to Section 301(c)(1) of the Act seeking widow’s benefits3

and, (6) whether the WCJ issued a reasoned decision. Because the first five issues relate to whether

Claimant met her burden under Section 301(c)(1) of the WC Act, they are all subsumed thereunder.

3

Under Section 307(2) of the Act, 77 P.S. § 561(2), widows are entitled to 51% of the

decedent’s wages, not to exceed the statewide average weekly wage. Claimant’s general eligibility

is undisputed. The WCJ found:

[Claimant], age 62 as of her testimony, is clearly [eli]gible to receive

benefits as a widow; no dispute exists as to her status. Here, there

was a forty-one (41)[-]year marriage (date of marriage, April 20,

1965) during which [Claimant and Bromley] lived together; they were

living together as of the date of [Bromley’s] death. [Claimant] is the

sole dependent entitled to benefits. Three children were born of the

marriage, but all are above the usual entitlement age, and none are

disabled; none were dependent on [Bromley] for support.

WCJ Dec. at 1.

2

due to Claimant’s work-related death. See C.R. Item 10. In all of the Petitions,

Claimant averred that Bromley’s date of injury/last date of employment was August

11, 2005. Employer denied the allegations in each of Claimant’s Petitions and raised

various affirmative defenses. See C.R. Items 3, 6, 12.

WCJ hearings were held on September 17, 2008, January 7, June 1 and

September 16, 2009, and January 20, April 19, July 26 and October 25, 2010. By

February 4, 2011 decision, the WCJ granted the Fatal Claim Petition and ordered

Employer to pay Claimant benefits based upon Bromley’s $1,738.08 average weekly

wage, commencing on June 23, 2006, the date of Bromley’s death (Original

Decision). The WCJ also directed Employer to pay 10% interest on all deferred WC

payments, $3,000.00 toward Bromley’s funeral expenses, plus litigation costs and

attorney’s fees. Employer appealed to the Board which, on August 9, 2013, held that,

although the WCJ found that Bromley’s death was due to work-related exposure to

carcinogens, since the WCJ did not state whether the conclusions were made pursuant

to Section 301(c)(1) or 301(c)(2) of the Act, the Board could not determine whether

substantial evidence supported the WCJ’s award. Accordingly, the Board remanded

the matter for the WCJ to specify whether he had awarded the benefits pursuant to

Section 301(c)(1) of the Act or under Section 301(c)(2) of the Act.4

On remand, the WCJ conducted additional hearings on March 7 and

September 10, 2014. On September 16, 2014, the WCJ reaffirmed his Original

Decision and added that “Claimant has met the burden of proof required under

Section 30[1](c)(1) of the Act, with benefits to [Claimant] being appropriately

4

Under Section 301(c)(2) of the Act, claimants who establish that certain occupational

diseases enumerated in Section 108 of the Act, added by Section 1 of the Act of October 17, 1972,

P.L. 930, 77 P.S. § 27.1, are entitled to a rebuttable presumption that the diseases are work-related

or “arose out of and in the course of [] employment[.]” Section 301(e) of the Act, 77 P.S. § 413.

Section 301(c)(1) of the Act does not allow for a similar presumption. See Pawlosky v. Workmen’s

Comp. Appeal Bd., 525 A.2d 1204 (Pa. 1987).

3

granted as entered in the [O]riginal Decision.” WCJ Remand Dec. at 3 (emphasis

added). The WCJ specifically held: “The instant matter meets the provisions of

Section 301(c)(1) [of the Act] as a repetitive/cumulative[-]type injury by way of

exposure to carcinogenic agents in the workplace over an extended period of

time resulting in bladder cancer and death[.]” WCJ Remand Dec. at 3 (emphasis

added). Employer appealed from the WCJ’s remand decision to the Board. On

March 30, 2016, the Board affirmed the WCJ’s remand decision. Employer appealed

to this Court.5

Initially, Section 301(c)(1) of the Act provides, in relevant part:

The terms ‘injury’ and ‘personal injury,’ as used in this

[A]ct, shall be construed to mean an injury to an employe,

regardless of his previous physical condition, . . . arising in

the course of his employment and related thereto, and

such disease or infection as naturally results from the

injury or is aggravated, reactivated or accelerated by the

injury; and wherever death is mentioned as a cause for

compensation under this [A]ct, it shall mean only death

resulting from such injury . . . , and occurring within

three hundred weeks after the injury.

77 P.S. § 411(1) (emphasis added). Accordingly, “[i]n a fatal claim proceeding, the

surviving family member bears the burden of proving that the decedent sustained an

injury in the course and scope of employment and that the decedent’s death was

causally related to the work-related injury.” J.D. Landscaping v. Workers’ Comp.

Appeal Bd. (Heffernan), 31 A.3d 1247, 1252 (Pa. Cmwlth. 2011).

“[T]he word ‘injury’ . . . is given no express statutory meaning” in

Section 301(c)(1) of the Act, and “does no more than state that an injury is an injury.”

5

“On review[,] this Court must determine whether constitutional rights were violated, errors

of law were committed, or necessary findings of fact were supported by substantial competent

evidence.” Stepp v. Workers’ Comp. Appeal Bd. (FairPoint Commc’ns, Inc.), 99 A.3d 598, 601 n.6

(Pa. Cmwlth. 2014).

Employer filed with this Court a request for supersedeas pending the appeal. By July 1,

2016 memorandum and order, this Court denied the supersedeas request.

4

Pawlosky v. Workmen’s Comp. Appeal Bd., 525 A.2d 1204, 1209 (Pa. 1987).

However, the term “has been broadly defined to encompass all work-related harm

including ‘any hurtful or damaging effect which may be suffered by anyone.’”

Jackson Twp. Volunteer Fire Co. v. Workmen’s Comp. Appeal Bd. (Wallet), 594 A.2d

826, 828 (Pa. Cmwlth. 1991) (emphasis added) (quoting Pawlosky, 525 A.2d at

1209). Thus, our courts have declared that an “injury” under Section 301(c)(1) of the

Act need not arise from an accident or specific physical bodily trauma, but can

include a disease not statutorily defined as an “occupational disease” under Section

301(c)(2) of the Act or The Pennsylvania Occupational Disease Act (the ODA),6 that

is caused by exposure to a job-related hazard. See Pawlosky; see also McCullough v.

Xerox Corp., 581 A.2d 961 (Pa. Super. 1990); Standard PA Practice 2d (2011) §§

167:220, 167:241. Specifically, “[b]ased on [the] Pawlosky[] [Court’s] broad

interpretation of ‘injury’ in [S]ection 301(c)(1) [of the Act], it is now well settled

that a claimant can establish a right to benefits for an ‘injury’ in the nature of a

work-related disease[,]” i.e., a disease as an injury claim.7 Brockway Pressed

6

Act of June 21, 1939, P.L.566, as amended, 77 P.S. §§ 1201-1603.

7

Accordingly, there is no support for Employer’s argument that bladder cancer “is not an

alleged injury” and “is not a condition that falls within the statutory meaning of” Section 301(c)(1)

of the Act, or that “it can only be classified as an occupational disease under [the ODA.].”

Employer Br. at 32.

When the ODA was enacted in 1939, occupational diseases were listed therein that were not

covered by the Act. However, in 1972, Sections 108 and 301(c) of the Act were amended to

include occupational diseases as compensable injuries. The amendments covered only disabilities

due to exposures after June 30, 1973, whereas the ODA remained in force for older claims. See

David B. Torrey & Andrew E. Greenberg, Workers’ Compensation: Law & Practice §§ 5:1, 5:2,

5:10 (3rd ed. 2008).

The [ODA] remains unrepealed. Obviously, one of the main reasons

for not repealing it was to make clear that the [ODA] was to remain in

force with respect to occupational diseases contracted prior to the

effective date of the 1972 disease provisions of the [Act]. It is worth

noting that the diseases covered by the [ODA] are essentially similar

to those provided for in [S]ection 108 of the [Act].

Pawlosky, 525 A.2d at 1210 n.9.

5

Metals v. Workers’ Comp. Appeal Bd. (Holben), 948 A.2d 232, 234 (Pa. Cmwlth.

2008) (emphasis added).

I. Whether Claimant met her burden under Section 301(c)(1) of the Act.

Employer argues that Claimant failed to meet her burden under Section

301(c)(1) of the Act of proving that Bromley’s death was caused by his exposure to

chemicals in the workplace within the 300 weeks preceding his death. We disagree.

In support of her Fatal Claim Petition, Claimant testified that, with the

exception of a broken leg in 1964, hernia surgery in 1993 and a diabetes diagnosis

approximately three years before his death,8 see R.R. at 400a, Bromley was “very

healthy” up to the time that he noticed blood in his urine in May or June 2005. R.R.

at 384a. Claimant recounted that, because Bromley’s physician thought he had a

Moreover, our Superior Court specifically stated that,

failure to meet the definition of occupational disease [under Section

301(c)(2) of the Act] does not exclude a claimant from compensation

under the [WC] system. See generally Pawlosky. . . . Such

definitions are merely procedural means of creating a non-

conclusive presumption that an injury was work-related. Failure to

establish the presumption [under Section 301(c)(2) of the Act] is not

dispositive; the claimant may still go forward in making out a case

for [WC benefits under Section 301(c)(1) of the Act] absent the

presumption.

McCullough v. Xerox Corp., 581 A.2d 961, 964 (Pa. Super. 1990) (emphasis added). Thus,

disability due to an occupational disease that is not specifically listed in the ODA or Section 108 of

the Act, 77 P.S. § 27.1, as incorporated by Section 301(c)(2) of the Act, may still be compensable,

as long as the requisite causation is established. Pawlosky.

Finally, Section 444 of the Act, added by Section 6 of the Act of October 17, 1972, P.L. 930,

provides, in pertinent part, that “any person may pursue, in the alternative, a claim for compensation

under this [A]ct and a claim for compensation under [the ODA].” 77 P.S. § 1000. Accordingly,

“[WC b]enefits for disabilities due to occupational diseases may be claimed under either [the ODA

or the Act,] or under both in the alternative.” Oscar Mayer & Co. v. Workmen’s Comp. Appeal Bd.,

425 A.2d 879, 879 (Pa. Cmwlth. 1981).

8

Claimant articulated that Bromley’s diabetes was controlled with diet and medication

(Glucophage). See R.R. at 384a, 399a, 405a.

6

prostate infection, Bromley was prescribed a six-week course of Cipro. See R.R. at

407a. Bromley underwent diagnostic testing of his bladder in June 2005, and surgery

to remove a tumor on August 11, 2005. See R.R. at 403a-404a.

Claimant explained that Bromley served his four-year electrician

apprenticeship at Sun Ship Building and Dry Dock (Sun Ship) in the 1960s. He

worked for Scott Paper’s foam division until approximately 1971. Thereafter, he was

employed at DuPont for approximately six months. In 1973, he returned to Scott

Paper at Employer’s Chester facility. See R.R. at 362a-364a.

Claimant related that Bromley worked at Employer’s plant until August

2005, but returned to work for Employer for an additional 40 hours in February 2006

in order to retire with seven paid vacation weeks. See R.R. at 400a-401a. She

testified that, although she did not have first-hand knowledge of Bromley’s working

conditions, she observed that the entire time he worked for Employer, “[h]is clothes

would have little holes in them,” and “he would have die [sic] on his hands when he

came home.” R.R. at 369a. She also recalled that there regularly was paper dust and

dye on his work clothes, the latter of which she could never get out. See R.R. at

370a-373a. Claimant could not specify how often Bromley came home with dye on

his hands or clothes, only that “[he] went to work, came home and sometimes he had

die [sic] on it and sometimes he did not,” but it happened frequently enough that she

just expected it. R.R. at 372a.

Further, Claimant described that Bromley smoked for approximately one

year, but he quit in 1965 when Claimant became pregnant, and he never smoked

thereafter. See R.R. at 392a-395a. She reported that although Bromley’s father

smoked at home while Bromley was growing up, Bromley’s mother required his

father to smoke outside. See R.R. at 391a, 395a-397a.

Robert Bonkowski (Bonkowski) testified that he has been an electrician

for Employer since 1971. See R.R. at 5a, 32a. He met Bromley during their

7

electrician apprenticeships at Sun Ship in the 1960s. See R.R at 46a. He related that

Bromley started the four-year program before he did, but they overlapped three of

those years, and they worked together at Employer’s facility from 1973 until Bromley

retired. See R.R. at 46a.

Bonkowski described that Employer’s facility consists of six major

buildings housing four types of operation areas – a powerhouse, pulp delivery, tissue

mill, and finishing/distribution – all of which he has worked in over the years. See

R.R. at 6a-7a. Bonkowski reported that the electricians were charged with keeping

the process running so, “[i]f it was part of the process, [they] worked on it.” R.R. at

24a; see also R.R. at 14a.

Bonkowski stated that Employer’s electricians initially worked from a

central shop and were assigned around the plant as needed, to regularly repair and test

machines, and to conduct “capital work” like new machine wiring, lighting,

construction, clean-up, etc.9 R.R. at 9a. Bonkowski specified that he and Bromley

were leaders from 1993 to 1997, meaning that they also purchased needed materials,

conducted safety meetings and created electrician job assignments. See R.R. at 8a,

11a. Moreover, Bonkowski recalled that when the central shop was dismantled in

1997 and the electricians were “assigned to individual assets” or families (i.e., given

specific area assignments), he was assigned to tissue mill 17 and Bromley was

assigned to the cogeneration facility (CCF) until 2000, when Bromley replaced the

napkins department electrician. R.R. at 8a; see also R.R. at 29a, 32a-33a, 43a.

Bonkowski articulated that, as electricians who worked throughout

Employer’s facility, he and Bromley came into contact with the various chemicals

used in Employer’s processing. See R.R. at 14a, 23a. He expounded:

9

Bonkowski testified that jobs were assigned by work order, horn call or shift supervisor’s

verbal request. See R.R. at 14a.

8

There was [sic] chemicals that were taken away from us

because they were called -- considered hazardous. One of

them was xylene, was a cleaning solvent that was used on

the machines. We were exposed to Tap Free that was taken

[] away from the mechanics [in the 1980s], because it was

considered dangerous.[10] Tazcon (ph) was a penetrating oil

that was removed, a lot of them just disappeared. I don’t

know if you can say they were removed, but I know our

cleaning tank, where we had a wash-up tank in the shop that

had the One-One-One in it, and I know that was removed.

Q. A cleaning solvent One-One-One?

A. Yeah. It had a code One-One-One on it. I don’t know

what the breakdown is but that was considered bad. We

were exposed to -- I guess we were exposed to

[polychlorinated biphenyl (]PCBs[)], which were replaced, I

guess, with number 10 transformer oil. PCBs that we used

to check test and work a lot with other vendors and stuff

like that. I mean, even the processes were changed, they

took kerosene out of the plant. They took formaldehyde out

of the plant [in the late 1980s]. They took milk of lime,

which was used for bleaching [recycled paper], which was

chlorine and lime and it was mixed, and it was called milk

of lime that was pumped out the plant, it was a bleach plant,

actually we had a bleach plant there [until the mid-1990s].

They had chlorine tanks, tanker trucks, train tanks, I guess

you would call them. That was full of chlorine.

Q. And you would come in contact with that as an

electrician?

A. Sure, because we had to disconnect motors, replace[]

rotted conduits, seal tights, the pipe fighters [sic] might be

pulling a pump. So in order to get the pump out, we had to

pull the motor out to make room for the new pump [to go]

in, and the mechanics would have to draw tap the motor

bases and things like that, so yeah.

Q. And when things would either disappear or be directly

pulled, were there ever any safety meetings with

management where that would be discussed?

10

Bonkowski clarified that although Employer no longer stocked Tap Free or Tap Magic,

the electricians all had cans of it that they continued to use thereafter until it was gone. See R.R. at

40a.

9

A. No, not really. Just went out and did your job.

Q. Is there any particular manager that would ever give you

any direction at all in safety, in dealings with these

chemicals that you recall?

A. I would say no, not really. Now, they did take asbestos

brakes out of the areas [in the early 1990s]. We did a lot of

asbestos brakes, transfer asbestos brakes, we used to take

care of the elevators. The freight elevators, they were all

asbestos brakes, and we would repair the motors in the shop

with asbestos brakes, blow out the motor and just blow

them out in the shop, and we didn’t have any special

HIPAA [sic] vacuum cleaner like they have today.

R.R. at 15a-17a; see also R.R. at 19a-23a, 42a.

Bonkowski described that, until the xylene tank was removed from the

plant, he and Bromley came into contact with xylene when they changed motor

pumps, and when it was sprayed on felts to clean them before starting the

manufacturing process. See R.R. at 18a. Bonkowski described that “the fumes were

so bad and they stunk so bad you’d be gagging and coughing, you’d have to leave the

area . . . .” R.R. at 18a. He recounted that Employer eventually installed an alarm for

people to leave areas where xylene was in use. See R.R. at 18a. He understood that

xylene was removed from the plant after the tank leaked into the well shop and

created an environmental hazard in approximately the late 1980s or early 1990s. See

R.R. at 17a, 19a, 39a-40a. Bonkowski recalled that, although he did not actually

witness Bromley with xylene on his clothes, all of the electricians were exposed, and

“[Bromley] being on shift longer . . . was probably exposed to it more[.]” R.R. at

19a.

Bonkowski also expressed that the electricians, including Bromley, were

exposed to significant amounts of industrial dyes, not only in the large tubs, and

through the dispensing hoses, but because dye was splashed everywhere, including on

motors, walls, doors, handrails, steps, starters and the floor, and they would touch it

10

and kneel in it to do their work, particularly in the CCF and napkins area. See R.R. at

23a-27a. Bonkowski specifically recalled that silica ash also covered every surface in

the CCF, including electrical panels, the dust system, walkways, doorways, floors and

even the road due to trucks hauling it to the landfill. See R.R. at 28a, 35a. He also

asserted that dust from culm (i.e., cheap coal) burned at Employer’s facility was “all

over the place” in the CCF. R.R. at 29a; see also R.R. at 30a.

Bonkowski recounted that, during the 2½ years that he and Bromley

worked the same shift:

A. Well, we worked on brakes together, we worked on

cranes together, because we were the two electricians

assigned to the main plant. So we were exposed to asbestos

dust on the cranes, elevators, brake dust. We pulled cable

and wire through the powerhouses and through the

basements, asbestos on top of the old pipes, when you pull

wire across the pipes the insulation was junky, so as you’re

wiring upon wire you’re cutting into the -- sawing into the

asbestos through the old basements and stuff, the old pipe

covering.

Q. Did you see any flaking or dust in that atmosphere?

A. Sure.

Q. Where else did you work with him?

A. Well, I mean, we’ve been all through the bleach plant

area. I mean, we’ve been through -- covered all the calls in

the bleach plant, the pulp prep department, in the beaters

and allies (ph), and things like that, that’s where kind of a

lot of the old pumps were resin pumps, stuff like that.

R.R. at 30a-31a.

Bonkowski acknowledged that, although he and Bromley worked in

separate areas between 1997 and 2000, and he did not know specifically what day-to-

day work Bromley did at that time, Bonkowski was aware of the conditions Bromley

was exposed to in the CCF during that time, “because [Bonkowski had] been over

11

there and [the ash was] everywhere.” R.R. at 35a; see also R.R. at 34a, 36a.

Bonkowski also knew that the napkins area where Bromley worked from 2000 until

2005 was approximately 80 by 300 feet in size and contained four machines, one of

which dyed the napkins, but since dye was splashed all over the area, Bromley would

have been in contact with it, whether he was working on the dye machine or not. See

R.R. at 44a, 52a-53a.

Bonkowski testified that Employer’s electricians were given plastic,

accordion-type dust masks to wear, but their use was emphasized only when

employees made motor brush changes or worked on brakes, due to carbon dust and

asbestos. See R.R. at 20a-21a. He also stated that protective clothing and leather

gloves were available to the electricians, but that no one really pushed their use or

warned that they should not enter a dye area without them, and when they did wear

the leather gloves they would get soaked with dye. See R.R. at 27a-28a, 41a.

Although he remembered Employer providing protective suits at some point, he does

not recollect when, and he explained that since they were not rubber, he could not say

whether they were water, dye or ink-repellant. See R.R. at 41a. In addition,

Bonkowski recalled Employer’s medical department fit-testing and assigning

respirators to the electricians approximately every 12 to 18 months, and the

electricians undergoing pulmonary function tests every three years as a condition of

their employment. See R.R. at 36a-37a. He pronounced that the respirators were

used primarily for ash rather than dye exposure. See R.R. at 28a, 37a-38a, 41a.

Bonkowski did not believe that he and Bromley were exposed to

asbestos or other chemicals during their Sun Ship apprenticeships because the ships

were empty steel hulls, and the apprentices were not informed that there was asbestos

present. See R.R. at 48a-49a. Employer, on the other hand, expressly notified

employees that there was asbestos at its facility, and pipes were so marked. See R.R.

12

at 48a. Bonkowski represented that he did not witness Bromley smoke during the

more than 40 years that he knew him. See R.R. at 49a-50a.

Jack Parris (Parris) testified that he has worked for Employer as an

electrician since 1969. See R.R. at 70a. He confirmed that electricians are

responsible for handling any electrical problem throughout Employer’s facility. See

R.R. at 70a. Parris represented that he has known Bromley since 1966, when they

worked at Sun Ship together. See R.R. at 70a, 97a. He recalled that he and Bromley

worked together for Employer from 1973 until 2006. See R.R. at 70a-71a. Parris

explained that since he worked swing-shift for the past 20 years, he and Bromley

worked together at least one-third of the time. See R.R. at 95a-97a, 104a. Parris

asserted that he and Bromley have had essentially the same job duties, and he

observed Bromley at work. See R.R. at 71a.

Parris described that he has been exposed to hazardous chemicals for

decades while working for Employer, including carbon dust from cranes and motors,

asbestos, xylene, bleach, formaldehyde, and dust and liquid dyes. See R.R. at 73a-

80a. He declared that he and Bromley were exposed to carbon dust while working on

cranes, brakes and motors, and asbestos throughout the facility. Parris specifically

recalled that xylene was sprayed freely from an 1½ inch hose to clean Employer’s

machines, which he and Bromley would smell and then experience headaches. See

R.R. at 75a-77a. He recounted that Employer eventually used an alarm to warn

employees to leave areas where xylene was being sprayed. See R.R. at 75a. Parris

articulated that he and Bromley while working together for Employer were exposed

for decades to bleach liquor, formaldehyde and asbestos. See R.R. at 76a-79a, 88a.

Parris further recollected a time when the electricians were told not to drink water

from the central shop fountain because, although it was clean enough to use for

processing, the city water may have been mixed with chemicals and sewage. See

R.R. at 87a-88a.

13

Regarding industrial dye exposure, Parris testified that he and Bromley

worked together in the several areas where the dyes were used. Parris described that

powder dyes were used in the pulp prep area:

[W]e had to go in there and change the motors out . . . [and]

work on the motor starters and . . . , you go like that,

(indicating) tap something you gotta watch because [there]

would be [powder dye] dust flying, so you knew you had to

be careful when you went in there. Never wore a respirator.

They never said wear a respirator . . . , and you’d get it on

your hands.

R.R. at 80a.

Parris explained that, although the liquid dye was more contained than

the powder dye, the electricians had to work on the pumps under the vats. See R.R. at

81a. He related that the liquid dye was all over the machine areas and the walls, and

he and Bromley, as the young guys, were sent there together because the older

electricians avoided the messy jobs. See R.R. at 82a-83a, 107a-108a. Parris recalled

that the electricians sometimes wore suits to keep from dirtying their clothes, but no

one ever told them not to touch the dye. See R.R. at 81a. He maintained that even

when the motors were brought to the electricians for service, they were covered in

dye that would get on their hands. See R.R. at 81a, 106a.

Parris stated that the electricians were not given respirators to work with

the dyes. See R.R. at 83a, 108a. He specified that employees were only trained to

wear their respirators in the CCF for protection from silicone. See R.R. at 83a-84a,

108a. Parris noted that he also used his respirator in areas like the coal yard due to

the dusty mist, but not if he was simply going there to flip a switch. See R.R. at 84a-

85a. He confirmed that the electricians were tested and fitted for personal respirators

every three years. See R.R. at 84a.

Parris also testified that he has worked around Employer’s asbestos-

covered pipes in the fan houses, attics, ceilings and the electrician’s central shop.

14

See R.R. at 85a. He represented that the 15 by 15 “motor rec room” where

electricians’ computers have been located for years, has the highest concentration of

asbestos, but since it is not flaking, Employer has yet to close it up. R.R. at 86a.

Parris confirmed that Bromley left shift work in the early 1990s,

Bromley and Bonkowski were leaders, Bromley was next assigned to CCF, then to

napkins until the equipment was sold and the department closed down in

approximately 2000 and Bromley removed the power. See R.R. at 89a-91a, 102a-

103a. He described that, as a leader between 1993 and 1997, Bromley did mostly

office work but, he would have done whatever electrical work was necessary when he

worked overtime. See R.R. at 91a-92a. Parris stated that Bromley worked in napkins

until the last machines were removed within the six months before Bromley left

Employer. See R.R. at 91a, 93a-94a, 111a. Even without the napkin machines,

Bromley was exposed to the remaining dye until his last day of work. See R.R. at

111a-112a. Parris concluded that Bromley was exposed to all of the same

contaminants that he was exposed to over their decades of working for Employer.

See R.R. at 88a. Parris admitted that he and Bromley smoked, but less than a pack a

day, and he recalled that Bromley quit more than 30 years earlier. See R.R. at 99a-

102a.

Claimant presented the October 19, 2009 and June 25, 2010 deposition

testimony of Barry L. Singer, M.D. (Dr. Singer). Dr. Singer explained that his

practice has focused primarily on oncology and hematology over the past 25 years,

only 2% to 3% of which has involved bladder cancer because “[i]t’s not one of the

most common cancers.” R.R. at 131a; see also R.R. at 134a. In preparation for

Bromley's evaluation, Dr. Singer reviewed Bromley’s medical records, including his

chest x-rays which reflected that Bromley had “asbestos lungs.” R.R. at 141a. He

also read Claimant’s, Parris’ and Bonkowski’s deposition testimony, Employer’s

material safety data sheets (MSDS), materials from the National Institute for

15

Occupational Safety and Health (NIOSH) and the International Agency for Research

on Cancer (IARC), and the independent medical evaluation report of Employer’s

oncology expert Alan J. Lippman, M.D. (Dr. Lippman). Dr. Singer stated that he also

reviewed Occupational Safety and Health Administration (OSHA) information, the

NIOSH carcinogen list and a KC Safety Tech Library Chemical Index. See R.R. at

165a-168a.

Dr. Singer described that any toxins or their byproducts that get into the

urine sit in the bladder and damage the transitional epithelium therein, which can lead

to metaplasia or dysplasia and, eventually, cancer. See R.R. at 140a-141a, 204a. Dr.

Singer declared that smoking is the prime cause for approximately 50% of bladder

cancer cases, due to benzene in cigarettes, but stated that 20% to 30% of bladder

cancers are caused by occupational exposure to chemicals. See R.R. at 140a, 142a-

143a, 163a-165a, 176a. He stated that workers who are not protected or who use

minimum protection are at a higher risk of occupational exposure, unless that work

area is 100% sealed. See R.R. at 176a-177a, 216a.

Dr. Singer produced abstracts of medical articles he reviewed that link

bladder cancer to chemical and asbestos exposure. See R.R. at 199a, 221a-231a,

244a-250a, 593a-622a. In particular, in Occupation and Bladder Cancer Among Men

in Western Europe (2003), higher bladder cancer risks were observed in electrical

workers and men employed in industrial chemical manufacturing. See R.R. at 244a.

In Bladder Cancer and Asbestos in Spain (1988), “the results [of a study conducted

between 1978 and 1982] showed that bladder cancer is associated with occupational

exposure to asbestos.” R.R. at 246a. The Italian study summarized in Occupation

and Risk of Bladder Cancer (1990), “confirm[ed] the well[-]known association

between bladder cancer risk and dyestuff production[.]” R.R. at 248a. In

Occupational Exposure to Chemical and Petrochemical Industries and Bladder

Cancer Risk in Four Western Canadian Provinces (2004), the study suggested that

16

there is an increased risk of bladder cancer in workers exposed to asbestos and

benzidene over the general population. See R.R. at 249a. In Occupational and Non-

Occupational Risk Factors in Bladder Cancer Patients in an Industrialized Area

Located in Former East-Germany (2005), bladder cancer was found to have been

overrepresented in patients with occupational exposure to asbestos and chlorinated

solvents. See R.R. at 250a.

Dr. Singer testified that Bromley’s work took him all over Employer’s

plant, and the portion of MSDS for chemicals used by Employer in areas where

Bromley worked since 1995 that he examined11 reflect that Bromley would have been

exposed to multiple carcinogenic compounds. Dr. Singer further explained:

Asbestos has some relationship to all cancers involving the

colon, the bladder, [and] the lung. So less so [sic] in the

bladder but certainly has potential to cause damage in

conjunction with other irritants.

As a sole exposure, it would be less likely but with [a]

combination of exposure with other compounds like

11

Dr. Singer clarified that he did not review all of the (approximately 250-300) MSDS that

he was given, because after he located approximately 15 which reflected carcinogenic compounds

used at Employer’s plant, he “felt it was sufficient.” R.R. at 206a; see also R.R. at 200a. Dr. Singer

identified the following carcinogenic compounds to which Bromley was exposed: ethyleneimine

(decorating dye; see 1997 request to approve 2-day trial in napkins - R.R. at 251a-254a), butadiene

(see 1997 MSDS for coupling grease - R.R. at 255a-264a), crystalline silica (see 1995 MSDS for

quick metal press fit, 1997 MSDS for CCF ash and a conditioning agent, 1999 MSDS for

permanent threadblocker, 2004 MSDS for pipe sealant - R.R. at 265a-289a, 333a-340a), nickel

compound (see 2004 MSDS for petroleum coke - R.R. at 290a-297a), toluene (see 1996 MSDS for

aerosol spray paint and 1999 MSDS for stencil ink spray - R.R. at 299a-303a, 316a-321a), xylene

(see 1996 MSDS for aerosol spray paint, 1996 MSDS for xylene and 2001 MSDS for metal and

textile markers - R.R. at 304a-309a, 316a-326a), ethylbenzene (see 1996 MSDS for xylene - R.R. at

322a-327a), trimethylbenzene (see 1996 MSDS for petroleum cleaner - R.R. at 310a-315a),

dicholorobenzidene (see 2000 MSDS for Bayprint yellow pigment - R.R. at 328a-332a), titanium

dioxide (see 1995 MSDS for quick metal press fit and 2001 MSDS for metal and textile markers -

R.R. at 304a-309a, 333a-340a), and trichloroethylene (see 2001 MSDS for lectra clean aerosol -

R.R. at 341a-344a). See R.R. at 157a, 201a-202a, 206a-215a, 217a, 220a, 251a-344a. He could not

determine from the MSDS whether, when or where Employer used those chemicals. See R.R. at

217a-218a.

17

benzene and the aniline dyes[, it] certainly would be a co-

contributing factor.

R.R. at 144a. Dr. Singer explained that the use of aniline dyes was prevalent in the

1970s and 1980s, and they were used by Employer, but they were mostly

discontinued by industries in approximately 1985 after they were known to be

carcinogenic. See R.R. at 146a, 162a-163a, 214a.

Dr. Singer did not believe that Bromley’s brief past smoking history

was significant in his developing bladder cancer because the benzene exposure was

“not long enough and the exposure was minimal.” R.R. at 141a. He did not consider

second-hand smoke exposure significant in Bromley’s case, since his wife did not

smoke, and his father smoked outside. See R.R. at 164a.

Dr. Singer represented that because bladder cancer generally occurs later

in life, and Bromley was only in his late 50s and did not smoke, his cancer was due to

“an intense exposure.” R.R. at 146a. He also deemed a significant sign of exposure

intensity that Bromley’s cancer was in the most advanced stage when it was

diagnosed, meaning that it had progressed rapidly and aggressively. See R.R. at

146a-147a.

Dr. Singer articulated that, based on the MSDS for the xylene Employer

used, the benzene compound ethylbenzene made up 15% of it. See R.R. at 143a,

150a. Dr. Singer reflected:

According to the reports I have read from the chemicals that

he was exposed to and the dyes and the silica and asbestos

that he was exposed to, [Bromley] had significant exposure

to chemicals and potential carcinogens or actual

carcinogens while at work at [Employer’s facility] and he,

according to [his] co-workers, he worked in areas that

certainly had exposure to xylene, previously to aniline dyes

and certainly he had exposure to asbestos because he had

asbestos lungs on [his] chest x-ray.

18

R.R. at 141a. He stated: “[Bromley’s] risk factors were . . . primarily the xylene,

which contained the 15% benzene, certainly the co-exposure to asbestos and silica,

especially the asbestos, [and] also some exposure that he had to the aniline dyes.

Those were the three main [sic].” R.R. at 146a. Dr. Singer could not point to any

scholarly journal article in which it was concluded that either asbestos, silica or

xylene alone could be a substantial factor in the development of bladder cancer. See

R.R. at 170a, 221a-231a. However, he concluded within a reasonable degree of

medical certainty that Bromley’s cumulative exposure to those chemicals at

Employer’s plant was a substantial contributing factor in his development of

bladder cancer and, thus, his death. See R.R. at 148a, 205a-206a, 221a.

Dr. Singer acknowledged that he did not know when or how much

xylene was used by Employer during Bromley’s employment, or whether Bromley

required medical attention for any acute exposures to xylene, silica or asbestos,

although it was clear that Bromley’s lungs revealed chronic changes due to asbestos

exposure and Claimant reported that Bromley experienced occasional rashes and

holes in his clothes.12 See R.R. at 153a-154a, 157a, 172a-173a, 213a, 218a-219a.

Employer presented the testimony of its environmental manager Gary

Baker (Baker).13 Baker described that he has been responsible for environmental

compliance, industrial hygiene accountability and testing at Employer’s Chester plant

since 1995. See R.R. at 417a-419a, 445a. He explained that, as chemical control

coordinator, he has had to review, approve and track all chemicals used in

Employer’s facility, and he inspects the site approximately once every six months.

12

Dr. Singer also admitted that Bromley’s medical records do not reference occupational

exposure to chemicals, but explained that he would not expect that there would be any such

references in oncology reports because the goal at that point is keeping the patient alive. See R.R. at

159a-161a, 175a, 218a-219a.

13

Baker was a safety, health and environmental coordinator at Employer’s corporate center

from 1990 to 1995.

19

See R.R. at 418a, 442a-443a, 465a. Baker presented MSDS for dyes used by

Employer, some of which date back to the late 1980s, and asbestos abatement and

removal records. See R.R. at 419a-420a, 437a, 444a, 451a, 460a. Baker reported that

he has Employer’s monthly chemical use inventory reports dating back only to 1995,

and they do not contain cleaning or maintenance chemicals, or oils and greases that

everyone uses. See R.R. at 464a-465a, 471a.

Baker articulated that since the CCF was built in the mid-1980s, it did not

contain asbestos. See R.R. at 420a-421a. He stated, however, that the manufacturing

facility does contain asbestos and, although a lot of it was removed in the 1980s and

early 1990s, there is still asbestos there, which Employer monitors daily. See R.R. at

421a-422a, 451a, 455a. Baker explained that although there is less asbestos now,

asbestos transite panels used for fireproofing walls remain in the facility. See R.R. at

455a, 459a. He added that there are also pipes covered in asbestos that are

encapsulated in aluminum, the ends are capped in material and the material is painted

over. See R.R. at 438a, 455a. Baker expressed that the asbestos could be exposed if

someone damages the steam line by cutting through the metal and into the insulation.

See R.R. at 439a. He claimed that electricians would normally be working on

electrical conduit that does not contain asbestos. See R.R. at 439a.

Baker related that when employees notify him of asbestos compromise on

the premises, an investigation is conducted regarding the cause and the exposure

potential then the compromise is either repaired or abated, and the air is monitored to

ensure there is no additional exposure risk. See R.R. at 424a-425a, 455a-458a, 459a.

Based upon his review of the results, Baker confirmed that, after asbestos repairs and

removals conducted at Employer’s facility, all actual and clearance testing fell below

permissible exposure levels.14 See R.R. at 425a-426a.

14

All asbestos clearance testing is conducted by an outside firm. See R.R. at 425a.

20

Baker disclosed that because there is the potential for employee exposure

to silica in the facility’s boiler 10 and CCF areas (including the office and water

treatment areas) due to anthracite coal burning, employees are required to wear

respirators in the CCF, particularly in the ash building. See R.R. at 426a-427a, 460a.

Baker agreed that Employer’s process was very different in 1970 than it

is now, and that he has no reason to dispute Bonkowski’s or Parris’ testimony about

xylene being sprayed to clean Employer’s mill prior to 1995. See R.R. at 449a. He

asserted that he has not approved or disapproved Employer’s use of xylene, and is not

aware of any xylene use at the facility since 1995. See R.R. at 429a. He recalled

that, since he has worked for Employer, an alarm is sounded to warn people to leave

the mill when the cleaning chemicals are being sprayed, but understands that has not

always been the case. See R.R. at 450a.

Baker claimed that Employer has only used kerosene to fuel small, five-

gallon, portable heaters used to heat certain areas of the plant during winter

shutdowns. See R.R. at 429a-430a. He stated that the heaters are not used in CCF,

since the machines generate their own heat. See R.R. at 430a. Baker recalled that

although the PCB transformers at Employer’s facility have been reduced because

they impose an environmental hazard, three still exist on-site, which are sealed and

regularly checked such that electricians and others who must work on them, are not

exposed to PCBs. See R.R. at 430a-431a, 461a-462a. He was not aware of any

employee reporting PCB exposure since 1995. See R.R. at 431a.

Baker reported that “[b]leach liquor refers to . . . the old style of mixing

two components together to make . . . concentrated sodium hypochlorite,” used by

Employer to whiten paper. R.R. at 431a. He confirmed that Employer still uses

sodium hypochlorite in the filter plant and in tissue manufacturing but, since at least

1995, a pre-mixed solution is shipped in. See R.R. at 431a, 462a, 471a-472a. He

reported that neither formaldehyde nor milk of lime have been used in the mill from

21

1995 to the present. See R.R. at 438a-440a. Although he recalled concerns being

expressed about the drinking water supplied by the Chester Water Authority, he

stated that Employer had it tested and confirmed that it “pretty much” matched

bottled water and, thus, was potable. R.R. at 433a.

Baker admitted that coal dust exists at Employer’s facility in the coal

yard, conveyor building and parts of the boiler building, and confirmed that CCF

personnel are exposed to the coal yard. See R.R. at 431a-432a. Baker acknowledged

that paper dust is generated in the paper mill areas of Employer’s facility, and where

parent rolls are converted to consumer-sized rolls, and some is evident in CCF or

where napkins are made. See R.R. at 433a.

Baker testified that he reviewed the MSDS for dyes and uncovered that

since the late 1980s only one – a bay print yellow – contained aniline and other

carcinogens, which was to be trialed for paper towels at Chester in approximately

2000 but, since it was not approved, it was never used. See R.R. at 434a-435a, 437a,

451a, 460a, 463a-464a. He recounted that powdered dyes had been used in the pulp

prep area of Employer’s plant to color tissues, but is not aware of powdered dyes

being used in the plant since 1995. See R.R. at 436a-437a.

Baker described that industrial dyes, inks, chemicals, silicas and asbestos

are still present at Employer’s facility, but that employees are trained on safe

chemical use, and are instructed regarding what safety equipment is needed for its

use. See R.R. at 468a, 470a. He stated that employees are required to report potential

chemical or other substance exposures to the medical and safety departments. See

R.R. at 424a, 433a-434a, 458a, 462a. He believed that the expectations are

represented in Employer’s safety handbook, which has been amended occasionally,

and that it falls under employee safety obligations to say no if they are asked to do

22

unsafe work, notify others that are doing unsafe work and report unsafe work.15 See

R.R. at 434a, 466a.

When asked whether there have been chemicals employees routinely

worked with since 1995 that were eventually deemed hazardous and removed from

use at the facility, Baker replied: “Nothing jumps to mind[.]” R.R. at 441a; see also

R.R. at 446a. He testified that Employer attempts to be proactive and remove

chemicals when it becomes known that they are dangerous. See R.R. at 467a-468a.

He recalled that he has made recommendations since 1995 to reduce environmental

impacts, like reducing volatile organic compounds and paper dust and silica

exposure. See R.R. at 446a-447a.

Baker recalled Bromley’s name but could not recollect his face, and he

admitted that he never directly supervised Bromley or even worked with him, nor did

he do the type of work Bromley did for Employer. See R.R. at 443a-444a.

Employer also presented Dr. Lippman’s April 7 and May 26, 2010

deposition testimony, which disagreed with Dr. Singer’s statements that Bromley had

significant exposure to chemicals and carcinogens during his employment. Dr.

Lippman further disputed Dr. Singer’s opinion that Bromley’s bladder cancer resulted

from Bromley’s exposure to a combination of chemicals, since there is no literature to

support such a conclusion. See R.R. at 503a. Rather, Dr. Lippman ultimately

concluded that Bromley would have suffered from bladder cancer whether he worked

at Employer’s facility or in a bookstore all of those years. See R.R. at 577a.

The law is well established that “[t]he WCJ is the ultimate factfinder and

has exclusive province over questions of credibility and evidentiary weight.” Univ. of

15

Baker described that employees undergo regular medical check-ups, and the respirators

are fit-tested to each employee. See R.R. at 428a-429a. He stated that, at least since 1995,

employees who do not wear the necessary respiratory protection are progressively disciplined with

counseling, verbal reprimands, written reprimands, suspensions and so on, depending upon the

number of violations. See R.R. at 427a.

23

Pa. v. Workers’ Comp. Appeal Bd. (Hicks), 16 A.3d 1225, 1229 n.8 (Pa. Cmwlth.

2011). “The WCJ, therefore, is free to accept or reject, in whole or in part, the

testimony of any witness, including medical witnesses.” Griffiths v. Workers’ Comp.

Appeal Bd. (Red Lobster), 760 A.2d 72, 76 (Pa. Cmwlth. 2000).

Based upon the evidence presented in the instant case, the WCJ granted

the Fatal Claim Petition because Claimant met her burden of proof under Section

301(c)(1) of the Act. In reaching his decision, the WCJ made the following relevant

credibility determinations:

3. On review, the evidence presented in support of this

claim is found credible and persuasive, and is accepted over

the defense evidence as there is a conflict. . . .

4. The testimony and opinions of Dr. Singer are found

credible and persuasive. His opinions are accepted over

those of Dr. Lippman as there is a conflict. The testimony

of both co-workers ([] Bonkowski and [] Parris), along

with the testimony of [Claimant], is found credible and

persuasive as their testimony, individually and

collectively, support the opinions of Dr. Singer.

....

9. [] Baker’s testimony would negate any significant

exposure since 1995; he did not however, dispute the

testimony of the two co-workers (e.g., the spraying of

xylene previously . . . .), and acknowledged the

continued presence of a number of compounds at the

facility, such as asbestos, paper dust, and coal dust, and

silica exposure . . . . Dr. Singer noted . . . that []

Bromley’s work environment subsequent to 1995

(within the last 300 weeks of [] Bromley’s employment;

he worked until August 2005) exposed him to ‘multiple

compounds that were carcinogens.’ . . . Dr. Singer

concluded that the occupational exposure resulted in

the bladder cancer. He testified: ‘ . . . the total exposure

that he had to all these chemicals was the cause of his

bladder cancer.’

24

10. Dr. Singer is well qualified to express relevant opinions

in this case. . . .

11. Dr. Lippman . . . totally negated the workplace as a

causal factor in [] Bromley’s bladder cancer. At the

same time, he did not seek to specifically identify the cause

of the bladder cancer. . . . Given the record of [Bromley’s]

work duties as an electrician, in various sections of the

plant, with associated exposure to carcinogen agents over

many years as described by his co-workers ([] Bonkowski

and [] Parris), the opinions of Dr. Singer are found more

reasonable and more credible than those of Dr.

Lippman on the issue of causation. This conclusion is

strengthened given Dr. Lippman’s inability to express any

specific opinion as to the cause of [Bromley’s] bladder

cancer (and even more so, considering that he

acknowledged that some 20 percent of bladder cancers are

generally accepted to be associated with specific

occupational exposures . . . ). Dr. Lippman’s . . . challenge

[to Dr. Singer’s opinions] goes to the weight to be given to

Dr. Singer’s opinions; the undersigned, in turn, has found

Dr. Singer’s testimony credible and persuasive, and as

noted the more reasonable and credible given the

circumstances presented by this record.

WCJ Dec. at 1, 3-4 (emphasis added).

Neither the Board nor the Court may reweigh the evidence or the WCJ’s

credibility determinations. Sell v. Workers’ Comp. Appeal Bd. (LNP Eng’g), 771

A.2d 1246 (Pa. 2001). Specifically, “Section 422(a) [of the Act, 77 P.S. § 834,] does

not permit a party to challenge or second-guess the WCJ’s reasons for credibility

determinations. [Thus, u]nless made arbitrarily or capriciously, a WCJ’s credibility

determinations will be upheld on appeal.”16 Pa. Uninsured Emp’rs Guar. Fund v.

Workers’ Comp. Appeal Bd. (Lyle), 91 A.3d 297, 303 (Pa. Cmwlth. 2014) (quoting

16

Capricious disregard “occurs only when the fact-finder deliberately ignores relevant,

competent evidence.” Williams v. Workers’ Comp. Appeal Bd. (USX Corp.-Fairless Works), 862

A.2d 137, 145 (Pa. Cmwlth. 2004). Capricious disregard, by definition, does not exist where, as

here, the WCJ expressly considered and rejected the evidence. Id.

25

Dorsey v. Workers’ Comp. Appeal Bd. (Crossing Constr. Co.), 893 A.2d 191, 195

(Pa. Cmwlth. 2006)).

On appeal after remand, the Board agreed that substantial evidence

supported the WCJ’s conclusion that Claimant met her burden under Section

301(c)(1) of the Act.17 In particular, the Board declared that “Dr. Singer’s testimony,

accepted as credible by the WCJ, is unequivocal in attributing [Bromley’s] bladder

cancer to workplace exposure primarily to xylene, with co-exposure to asbestos and

silica and some exposure to aniline dyes.” Board Op. at 15.

“As with all claim petitions, the elements necessary to support [a fatal

claim petition] award must be established by substantial evidence. Substantial

evidence has been defined as such relevant evidence as a reasonable mind might

accept as adequate to support a conclusion.” Gibson v. Workers’ Comp. Appeal Bd.

(Armco Stainless & Alloy Prods.), 861 A.2d 938, 943 (Pa. 2004). Here, although it is

undisputed that Bromley’s death was due to bladder cancer, the parties do not agree

that Bromley’s bladder cancer was caused by workplace exposure within the 300

weeks before his death. Employer specifically contends that the lay witness

testimony was insufficient to prove Bromley’s alleged workplace exposure, and that

Dr. Singer’s opinions on causation were based upon an inadequate factual record and

lacked scientific or medical basis.

17

We agree with Employer that, in Finding of Fact 11, the WCJ appears to have implied that

Employer had a burden to demonstrate an alternative cause for Bromley’s cancer (i.e., “[Dr.

Lippman] did not seek to specifically identify the cause of the bladder cancer.” WCJ Dec. at 3; “Dr.

Lippman’s inability to express any specific opinion as to the cause of Claimant’s bladder cancer[.]”

WCJ Dec. at 4). Claimant here had “a nevershifting burden of proving not only that [Bromley’s]

injury arose in the course of his employment but also that the injury was related to it.” Pawlosky,

525 A.2d at 1211 (emphasis added). However, since other substantial evidence supported the

WCJ’s conclusion, those references in the WCJ’s finding constituted harmless error.

26

a. Three Hundred-Week Calculation.

In order for a fatal claim to be compensable under Section 301(c)(1) of

the Act, an employee’s death must occur “within three hundred weeks[18] after the

injury.” 77 P.S. § 411(1) (emphasis added). Accordingly, “[t]his Court has

consistently held, without exception, that Section 301(c)(1) [of the Act] denies

benefits to a claimant when more than 300 weeks have elapsed between the

commencement of the compensable injury and the injury-related death.” Whitesell

v. Workers’ Comp. Appeal Bd. (Staples, Inc.), 74 A.3d 297, 300 (Pa. Cmwlth. 2013)

(emphasis added); see also Olsen Bodies, Inc. v. Workmen’s Comp. Appeal Bd.

(Gavas), 573 A.2d 238, 240 (Pa. Cmwlth. 1990) (“there is no recovery for death

occurring more than three hundred weeks after a non-occupational disease[-]type

injury”).19 However, “[i]t is well settled that for an injury to be compensable under

the Act, it is not required that the injury resulted from any sudden occurrence or

accident; it may be due to daily trauma . . . .” Pittsburgh Steelers Sports, Inc. v.

Workers’ Comp. Appeal Bd. (Williams), 814 A.2d 788, 793 (Pa. Cmwlth. 2002)

(emphasis added).

The parties do not cite and our research has not disclosed any reported

judicial decision that defines precisely when the “injury” occurs in disease as injury

cases under Section 301(c)(1) of the Act. However, David B. Torrey and Andrew E.

Greenberg, in Workers’ Compensation: Law & Practice (3rd ed. 2008), opined: “The

commencement date applicable to a disease as injury case [under Section 301(c)(1) of

the Act] is appropriately conceived of as the last date of injurious exposure to the

agent causing the disease, whether or not such last exposure is disabling.” Id. § 5:19

18

“Three hundred weeks is the equivalent of five years, nine months and one week.” City of

McKeesport v. Workers’ Comp. Appeal Bd. (Miletti), 746 A.2d 87, 89 n.5 (Pa. 2000). The date 300

weeks prior to Bromley’s June 23, 2006 death was September 22, 2000.

19

The Pennsylvania Supreme Court has since stated: “[N]owhere in the Act is there any

such phrase as ‘occupational disease-like.’ An ailment is either an occupational disease or it is not.

The quoted phrase is meaningless, and it causes confusion.” Pawlosky, 525 A.2d at 1210 n.8.

27

(emphasis added). Specifically, for death claims in disease as injury cases, Torrey

and Greenberg declared that if “the employee dies more than 300 weeks after the

injury (last injurious exposure to the hazardous condition), then the fatal claim will be

barred.” Id. § 5:20.

Further, in Kuo-Hom Hsu v. Workers’ Compensation Appeal Board

(Rohm & Haas) (Pa. Cmwlth. No. 328 C.D. 2013, filed October 15, 2013), this Court

declared that the Board properly required the claimant to

prove [for purposes of Section 301(c)(1) of the Act] that

[the d]ecedent was exposed to a hazard that caused his

brain cancer or injury within 300 weeks of his July 3,

2007 death, . . . because, in a death claim, Section

301(c)(1) [of the Act] requires that death occur within three

hundred weeks of the injury. 77 P.S. § 411(1).

Slip op. at 3 (emphasis added).20 Thus, this Court has determined for a fatal disease

as injury claim to be compensable under Section 301(c) of the Act, the employee’s

hazardous exposure is the injury from which the 300 week look-back period must

be calculated.

Here, the WCJ concluded on remand that

the instant matter meets the provisions of Section 301(c)(1)

[of the Act] as a repetitive/cumulative[-]type injury by way

of exposure to carcinogenic agents in the workplace over an

extended period of time resulting in bladder cancer and

death -- as other repetitive/cumulative[-]type injuries to the

muscles/nerves of the body have been found compensable

under this provision of the Act.

WCJ Remand Dec. at 3. Indeed, this Court has consistently held that “[a]n injury

[under Section 301(c)(1) of the Act] is compensable when it develops over a period

20

We acknowledge that this Court’s unreported memorandum opinions may be cited “for

[their] persuasive value, but not as a binding precedent.” Section 414 of the Commonwealth

Court’s Internal Operating Procedures, 210 Pa. Code § 69.414. In light of the nearly identical facts

of this more recently-decided case, it is cited herein for its persuasive value.

28

of time and results from a number of work activities in which the employee

engaged.” Curran v. Workmen’s Comp. Appeal Bd. (Maxwell Indus.), 664 A.2d 667,

670 (Pa. Cmwlth. 1995). In such cases, “determinations of the date of injury depend

largely on the facts of each case, the purpose for which the injury date must be

established, and the medical evidence presented.”21 Id. at 671.

Based upon the foregoing, we hold that, in order for Claimant’s claim to

be compensable under Section 301(c)(1) of the Act, she had to prove by substantial

evidence that Bromley’s last exposure to a hazard occurred on or after

September 22, 2000.22

21

“In cumulative trauma cases [under Section 301(c)(1) of the Act], the last day of

employment is generally used as the date of injury for purposes of notice and filing limitations.”

Meenan Oil Co., L.P. v. Workers’ Comp. Appeal Bd. (Pownall), 846 A.2d 793, 795 n.5 (Pa.

Cmwlth. 2004) (emphasis added); see also City of Phila. v. Workers’ Comp. Appeal Bd. (Williams),

851 A.2d 838 (Pa. 2004).

22

Our conclusion is consistent with proofs required for occupational diseases under Section

301(c)(2) of the Act. Thereunder, if an employee did not file a lifetime benefit claim, his death due

to a statutorily-defined occupational disease must occur “within three hundred weeks after the last

date of employment in an occupation or industry to which he was exposed to hazards of such

disease[.]” 77 P.S. § 411(2); Ingram v. Workers’ Comp. Appeal Bd. (Ford Elecs. & Refrigeration

Corp.), 940 A.2d 544 (Pa. Cmwlth. 2007).

The Pennsylvania Supreme Court has declared:

Although Section 301(c)(2) [of the Act] references the employee’s

‘last date of employment,’ 77 P.S. § 411(2), . . . the 300-week period

begins on the last day of employment-based exposure to the

hazard. See Sporio v. [Workmen’s Comp. Appeal Bd.] (Songer

Constr.), . . . 717 A.2d 525 [] ([Pa.] 1998); Cable v. [Workmen’s

Comp. Appeal Bd.] (Gulf Oil/Chevron USA), . . . 664 A.2d 1349 []

([Pa.] 1995) (plurality).

Tooey v. AK Steel Corp., 81 A.3d 851, 870 n.6 (Pa. 2013) (emphasis added); see also Farr v.

Workers’ Comp. Appeal Bd. (TRW, Inc.), 823 A.2d 1043, 1046 (Pa. Cmwlth. 2003) (“[t]he three-

hundred week period prescribed in [Section 301(c)(2) of] the Act is measured from the last date of

exposure to the hazard alleged to cause the disease, not from the last date of employment”).

29

b. Whether a workplace hazard existed to which Bromley was exposed on

or after September 22, 2000.

Relative to WC claims brought pursuant to Section 301(c)(1) of the Act,

this Court has declared:

[W]hether a hazard exists is a question of fact for the

[WCJ] to determine. Furthermore, . . . a claimant’s

burden of proof related to this issue is not overly

demanding. We have also asserted that ‘[s]ince claimant’s

exposure is a factual question, the claimant need not

present scientific evidence or expert testimony to prove

the existence of the hazard in the workplace.’ Mauger [&]

Co[.] v. Workmen’s Comp[.] Appeal B[d.] (Waltz), . . . 598

A.2d 1035, 1037 ([Pa. Cmwlth.] 1991). ‘The [WCJ] may

rely solely on the testimony of the claimant or other

witnesses to prove the existence of and exposure to the

hazard.’ Id.

Gray v. Workmen’s Comp. Appeal Bd. (Pittsburgh Bd. of Educ.), 657 A.2d 77, 80-81

(Pa. Cmwlth. 1995) (emphasis added).

As to proof of the existence of the disease-causing element

in the work environment, the courts have accepted lay

testimony, as well as other expert testimony, to support a

finding that the disease-causing element is present.

However, the testimony of a lay person appears to require

testimony of personal experience with the illness-causing

element and personal knowledge.

Craftex Mills, Inc. of PA v. Workers’ Comp. Appeal Bd. (Markowicz), 901 A.2d 1077,

1080-81 (Pa. Cmwlth. 2006) (emphasis added). Accordingly, lay testimony of first-

hand knowledge of a hazard gained from practical experience can be sufficient

to prove the existence of and exposure thereto. Gibson.

Because the WCJ did not have the benefit of Bromley’s personal

testimony regarding his specific workplace exposures to hazardous chemicals, the

law permitted the WCJ to consider and rely upon first-hand lay testimony regarding

30

Bromley’s work environment.23 Claimant testified regarding her personal knowledge

that over the years that Bromley worked for Employer, his clothing contained small

holes, and was covered in paper dust and dye.

Bonkowski and Parris worked with Bromley as electricians in

Employer’s Chester plant from 1973 until 2005, they had essentially the same job

duties and they observed Bromley at work. Employer’s electricians were charged

with keeping Employer’s entire process running, so they were directly exposed to all

areas of the plant, in, under and around the machinery, pipes and elevators.

Bonkowski testified from his personal experience that he and Bromley were regularly

exposed to industrial oils, bleach, formaldehyde, PCBs, kerosene, milk of lime,

bleach, coal ash, silica ash, carbon dust asbestos, and significant amounts of xylene

liquid and fumes and industrial dyes over the years since 1973, because coal and

silica dust and dyes covered the surfaces of Employer’s facility, and that asbestos

flakes and dust could be seen in the air. Bonkowski also specifically recalled seeing

Bromley with xylene-soaked pants on at least one occasion. Although several of the

chemical hazards were abated, and Bonkowski and Parris worked more closely with

Bromley before he was assigned to CCF in 1997 and the napkins after 2000, they

were aware that Bromley continued to be exposed to coal dust, silica ash and

23

Employer relies upon Gibson and May Department Stores v. Workmen’s Compensation

Appeal Board (Smith), 525 A.2d 33 (Pa. Cmwlth. 1987), to argue that the conclusory lay testimony

offered in this case was insufficient to support the WCJ’s findings. In Gibson, the co-worker’s lay

testimony was deemed insufficient to establish the decedents’ workplace asbestos exposure where

the co-worker “simply testified that he saw [the decedent] near a dusty, cottony material that [the

co-worker] was unable to identify.” Id. at 484. In May, this Court rejected decedent’s doctor’s

conclusion that the decedent contracted Legionnaire’s disease at work, because it was based solely

on the doctor’s assumptions that since the decedent worked with air-conditioning units, he was

exposed to damp environments, and that people who work in damp environments seem to be more

susceptible to Legionnaire’s disease than the normal population. Bonkowski’s and Parris’

testimony of first-hand experience with specifically-described chemicals and their personal

observations of Bromley’s exposure to them makes this case factually distinguishable from Gibson

and May.

31

industrial dyes in CCF between 1997 and 2000, and significant amounts of dye in

napkins from 2000 until he retired.

Baker admitted that Employer’s asbestos abatement records and his

monthly chemical inventory reports are limited in time, and that the latter do not

include the cleaning or maintenance chemicals or oils and greases employees

commonly used at the facility. Moreover, since Baker’s deposition was conducted

before Employer produced the MSDS for CCF and napkins from 1997 through 2005

which reflected that Employer continued to use hazardous chemicals in areas where

Bromley was assigned (including xylene containing benzene, despite Baker’s

representation to the contrary), Baker did not address them. Baker’s testimony did

not in any way contradict or refute Bonkowski’s or Parris’ testimony about the

continued use of hazardous chemicals or their specific presence in CCF and napkins

until 2005. Moreover, Baker confirmed that Employer’s employees, including

Bromley, during the relevant time period were exposed to silica dust and coal ash,

particularly in CCF, to dyes particularly in napkins, and asbestos throughout

Employer’s premises.

This Court has held:

‘In performing a substantial evidence analysis, this [C]ourt

must view the evidence in a light most favorable to the

party who prevailed before the factfinder.’ ‘Moreover, we

are to draw all reasonable inferences which are deducible

from the evidence in support of the factfinder’s decision in

favor of that prevailing party.’ It does not matter if there is

evidence in the record supporting findings contrary to those

made by the WCJ; the pertinent inquiry is whether the

evidence supports the WCJ’s findings.

3D Trucking Co., Inc., v. Workers’ Comp. Appeal Bd. (Fine & Anthony Holdings

Int’l), 921 A.2d 1281, 1288 (Pa. Cmwlth. 2007) (quoting Waldameer Park, Inc. v.

32

Workers’ Comp. Appeal Bd. (Morrison), 819 A.2d 164, 168 (Pa. Cmwlth. 2003))

(citations omitted).

The WCJ made specific findings that the testimony offered by Claimant,

Bonkowski and Parris was credible. Baker’s testimony did not refute that Bromley

was exposed to chemicals while working for Employer after September 22, 2000.

See WCJ Dec. at 1, Finding of Fact 4; see also Finding of Fact 9. Although a

significant amount of Claimant’s hazardous exposure evidence established that

Bromley was regularly exposed to harmful substances throughout Employer’s

premises between 1973 and 2000, viewing the evidence in Claimant’s favor in this

case, as we must, we hold that the undisputed lay testimony offered by Bonkowski,

Parris and even Baker constituted substantial evidence to support the WCJ’s findings

of Bromley’s exposure to asbestos, silica dust, xylene and dyes in Employer’s

workplace on or after September 22, 2000.24

c. Whether Bromley’s workplace hazard exposure was a substantial

contributing cause of his bladder cancer.

“[I]n the case of a fatal claim petition, [the surviving family member has

the burden to prove] that th[e] injury or disease was a substantial contributing

cause in bringing about the death of th[e] employee.” Gibson, 861 A.2d at 943

(emphasis added). “If the causal connection is not obvious, the connection must be

established by unequivocal medical testimony.” Dietz v. Workers’ Comp. Appeal Bd.

(Lower Bucks Cnty. Joint Mun. Auth.), 126 A.3d 1025, 1030 (Pa. Cmwlth. 2015).

“[M]edical testimony is unequivocal if a medical expert testifies, after providing

foundation for the testimony, that, in his professional opinion, he believes or thinks a

24

Bromley’s exposure on Employer’s premises during the 40 hours he returned to work in

February 2006 is not clear.

33

fact exists.”25 Amandeo v. Workers’ Comp. Appeal Bd. (Conagra Foods), 37 A.3d

72, 80 (Pa. Cmwlth. 2012) (quoting O’Neill v. Workers’ Comp. Appeal Bd. (News

Corp., Ltd.), 29 A.3d 50, 58 (Pa. Cmwlth. 2011)).

Dr. Singer received his medical degree from Johns Hopkins University

and is board-certified in internal medicine. He has specialized in oncology (75% of

his current practice) since the 1970s, and he has been involved with the treatment of

bladder cancer over his 40 years of practice. See R.R. at 126a-134a. Based upon his

experience and extensive review of Bromley’s medical records, the depositions,

Employer’s MSDS, NIOSH, OSHA and IARC literature and journal articles, he

concluded that Bromley’s co-exposure to xylene, asbestos, silica and dyes over the

years while working at Employer’s facility since 1973 until August 11, 2005 was the

substantial cause of the bladder cancer from which Bromley died.26

25

“The question of whether expert medical testimony is unequivocal, and, thus, competent

evidence to support factual determinations is a question of law subject to our review.” Amandeo v.

Workers’ Comp. Appeal Bd. (Conagra Foods), 37 A.3d 72, 80 (Pa. Cmwlth. 2012).

26

Employer argues that the WCJ erred by overruling Employer’s objection that Dr. Singer’s

June 25, 2010 testimony of a definitive link between asbestos and bladder cancer lacked foundation

because Dr. Singer “never established any scientific facts or data based on his own personal

knowledge or from any other source[.]” Employer Br. at 53.

“It is well settled that the admission of evidence is within the sound discretion of the WCJ.”

Washington v. Workers’ Comp. Appeal Bd. (State Police), 11 A.3d 48, 59 (Pa. Cmwlth. 2011). “[A]

WCJ’s determination regarding the admission of evidence will not be overturned without a showing

of an abuse of that discretion.” Id. Further,

[t]he law provides that

expert testimony is incompetent if it lacks an adequate basis

in fact. While an expert’s opinion need not be based on

absolute certainty, an opinion based on mere possibilities is

not competent evidence. This means that expert testimony

cannot be based solely upon conjecture or surmise. Rather,

an expert’s assumptions must be based upon such facts as

the jury would be warranted in finding from the

evidence. Accordingly, the Pennsylvania Rules of Evidence

prescribe a threshold for admission of expert testimony

dependent upon the extent to which the expert’s opinion is

based on facts and data:

34

“As this Court has stated on numerous occasions, it is within the

[WCJ’s] power to determine which medical witness he or she accepts as credible, in

whole or in part.” Mauger & Co. v. Workmen’s Comp. Appeal Bd. (Waltz), 598 A.2d

1035, 1041 (Pa. Cmwlth. 1991).

The fact that [Dr. Singer’s] opinion conflicted with those of

[Dr. Lippman] does not make it equivocal. As long as [Dr.

Singer] could state, with a reasonable degree of medical

certainty, that . . . [Bromley’s] condition was caused by his

total and cumulative exposures . . . at Employer’s place of

business . . . , Claimant provided substantial competent

evidence to support a finding of causation.

McGraw Edison Power Sys. v. Workmen’s Comp. Appeal Bd. (Kuzior), 561 A.2d

1327, 1330 (Pa. Cmwlth. 1989). Here,

[t]he record discloses that [Dr. Singer] testified with a

reasonable degree of medical certainty that [Bromley’s

bladder cancer] resulted directly from [co-]exposure to

[xylene, asbestos, silica and dyes]. Notwithstanding

Rule 703. Bases of opinion testimony by experts

The facts or data in the particular case upon which an

expert bases an opinion or inference may be those

perceived by or made known to the expert at or before

the hearing. If of a type reasonably relied upon by

experts in the particular field in forming opinions or

inferences upon the subject, the facts or data need not

be admissible in evidence.

Pa.R.E. 703.

Helpin v. Trustees of Univ[.] of P[a.], 969 A.2d 601, 617 (Pa. Super.

2009) (citation and quotation marks omitted).

Gillingham v. Consol Energy, Inc., 51 A.3d 841, 849 (Pa. Super. 2012) (emphasis added). Where,

as here, Dr. Singer’s opinions were based upon his experience and extensive review of Bromley’s

medical records, the depositions, Employer’s MSDS, NIOSH, OSHA and IARC literature and

journal articles, there was a foundation for a link between asbestos and bladder cancer and, thus, the

WCJ did not abuse his discretion by overruling Employer’s objection. See R.R. at 199a; WCJ Dec.

at 2.

35

[Employer’s] artful cross-examination to induce testimony

on the possibility of other etiologies, [Dr. Singer] never

recanted h[is] initial assertion of causation and, in fact,

reiterated on cross-examination that [Bromley’s co-

]exposure to [xylene, asbestos, silica and dyes] was the []

explanation for the disease.

Superior Tube Co. v. Workmen’s Comp. Appeal Bd. (Unger), 572 A.2d 258, 260 (Pa.

Cmwlth. 1990) (footnote omitted).

Here, the WCJ made specific findings that the testimony offered by

Claimant, Bonkowski and Parris was credible, and that such evidence individually

and collectively supported Dr. Singer’s credited testimony that Bromley’s workplace

hazard exposure was a substantial contributing cause of his bladder cancer. See WCJ

Dec. at 1, Finding of Fact 4; see also Finding of Fact 9. Accordingly, we conclude

that the WCJ’s findings of fact are supported by substantial record evidence.

Because there is substantial record evidence to support the WCJ’s

findings that Bromley sustained an injury in the course and scope of employment that

caused his death within 300 weeks of his last exposure, the WCJ properly determined

that “Claimant has met the burden of proof required under Section 30[1](c)(1) of

the Act[.]” WCJ Remand Dec. at 3 (emphasis added).

II. Whether the WCJ issued a reasoned decision.

Employer also argues that the WCJ’s decision was not reasoned

“because the WCJ found that Claimant met her burden of proof on her Fatal Claim

Petition, which [Employer] respectfully submits that she did not,” and “because, in

granting Claimant’s Fatal Claim Petition, the WCJ relied upon insufficient evidence

to reach his decision” and, finally, “because the WCJ merely adopted Claimant’s

Proposed Findings of Fact.” Employer Br. at 50-51. We disagree.

36

Section 422(a) of the Act[27] requires a WCJ to issue a

decision that permits an appellate court to exercise adequate

appellate review. In order to satisfy this standard, a WCJ

does not need to discuss every detail of the evidence in the

record. Rather, Section 422(a) of the Act requires WCJs to

issue reasoned decisions so that this Court does not have to

‘imagine’ the reasons why a WCJ finds that the conflicting

testimony of one witness was more credible than the

testimony of another witness.

Although our Supreme Court has held that a WCJ need not

explain credibility determinations relating to a witness who

testifies before the WCJ, Section 422(a) of the Act requires

some explanation of credibility determinations by a WCJ

with regard to conflicting deposition testimony in order to

enable this Court to review a WCJ’s decision. Under

Section 422(a) of the Act, a WCJ must articulate the

objective rationale underlying his credibility determinations

where the testimony of such witnesses is conflicting. A

WCJ may satisfy the reasoned decision requirement if

he summarizes the witnesses’ testimony ‘and adequately

explains his credibility determinations.’ Clear Channel

Broad. v. Workers’ Comp. Appeal Bd. (Perry), 938 A.2d

1150, 1157 (Pa. Cmwlth. 2007). Thus, while summaries of

testimony alone would be insufficient to satisfy the

reasoned decision requirement, where a WCJ summarizes

testimony and also objectively explains his credibility

determinations, the decision will satisfy the requirement.

Further, other evidence in the record may provide the

objective support necessary under Section 422(a) of the Act

for adequate credibility determinations.

Amandto, 37 A.3d at 76 (citations omitted; emphasis added).

Here, because the WCJ clearly and extensively summarized the

testimony and objectively explained his credibility determinations, we hold that the

WCJ issued a reasoned decision in accordance with Section 422(a) of the Act.

Based upon the foregoing, the Board’s order is affirmed.

___________________________

ANNE E. COVEY, Judge

27

77 P.S. § 834.

37

IN THE COMMONWEALTH COURT OF PENNSYLVANIA

Kimberly Clark Corporation, :

Petitioner :

:

v. :

:

Workers’ Compensation Appeal :

Board (Bromley), : No. 656 C.D. 2016

Respondent :

ORDER

AND NOW, this 4th day of May, 2017, the Workers’ Compensation

Appeal Board’s March 30, 2016 order is affirmed.

___________________________

ANNE E. COVEY, Judge

This is a copy of a public record, reproduced as it was published. It is not legal advice, and it may not be the version a court would rely on. Check the official source before you cite it.

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